JOHN LYDON to Endometriosis
This is a "connection" page, showing publications JOHN LYDON has written about Endometriosis.
Connection Strength
1.416
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Early growth response 1 transcription factor is essential for the pathogenic properties of human endometriotic epithelial cells. Reproduction. 2022 08 01; 164(2):41-54.
Score: 0.621
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A GREB1-steroid receptor feedforward mechanism governs differential GREB1 action in endometrial function and endometriosis. Nat Commun. 2024 Mar 02; 15(1):1947.
Score: 0.174
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CFP1 governs uterine epigenetic landscapes to intervene in progesterone responses for uterine physiology and suppression of endometriosis. Nat Commun. 2023 06 03; 14(1):3220.
Score: 0.166
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Endometrial Expression of Steroidogenic Factor 1 Promotes Cystic Glandular Morphogenesis. Mol Endocrinol. 2016 May; 30(5):518-32.
Score: 0.101
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Estrogen Receptor ? Modulates Apoptosis Complexes and the Inflammasome to Drive the Pathogenesis of Endometriosis. Cell. 2015 Nov 05; 163(4):960-74.
Score: 0.098
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Activated AKT pathway promotes establishment of endometriosis. Endocrinology. 2014 May; 155(5):1921-30.
Score: 0.087
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A new isoform of steroid receptor coactivator-1 is crucial for pathogenic progression of endometriosis. Nat Med. 2012 Jul; 18(7):1102-11.
Score: 0.078
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Intact progesterone receptors are essential to counteract the proliferative effect of estradiol in a genetically engineered mouse model of endometriosis. Fertil Steril. 2004 Sep; 82(3):673-8.
Score: 0.045
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ARID1A Is Essential for Endometrial Function during Early Pregnancy. PLoS Genet. 2015 Sep; 11(9):e1005537.
Score: 0.024
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The epidermal growth factor receptor critically regulates endometrial function during early pregnancy. PLoS Genet. 2014 Jun; 10(6):e1004451.
Score: 0.022