Connection

DAVID NELSON to Signal Transduction

This is a "connection" page, showing publications DAVID NELSON has written about Signal Transduction.
Connection Strength

0.139
  1. NEMO, NFkappaB signaling and incontinentia pigmenti. Curr Opin Genet Dev. 2006 Jun; 16(3):282-8.
    View in: PubMed
    Score: 0.077
  2. Intercepting IRE1 kinase-FMRP signaling prevents atherosclerosis progression. EMBO Mol Med. 2022 04 07; 14(4):e15344.
    View in: PubMed
    Score: 0.014
  3. Deletion of Fmr1 from Forebrain Excitatory Neurons Triggers Abnormal Cellular, EEG, and Behavioral Phenotypes in the Auditory Cortex of a Mouse Model of Fragile X Syndrome. Cereb Cortex. 2020 03 14; 30(3):969-988.
    View in: PubMed
    Score: 0.013
  4. MBD5 haploinsufficiency is associated with sleep disturbance and disrupts circadian pathways common to Smith-Magenis and fragile X syndromes. Eur J Hum Genet. 2015 Jun; 23(6):781-9.
    View in: PubMed
    Score: 0.009
  5. The Ectocarpus genome and the independent evolution of multicellularity in brown algae. Nature. 2010 Jun 03; 465(7298):617-21.
    View in: PubMed
    Score: 0.006
  6. The Physcomitrella genome reveals evolutionary insights into the conquest of land by plants. Science. 2008 Jan 04; 319(5859):64-9.
    View in: PubMed
    Score: 0.005
  7. Secretin receptor-deficient mice exhibit impaired synaptic plasticity and social behavior. Hum Mol Genet. 2006 Nov 01; 15(21):3241-50.
    View in: PubMed
    Score: 0.005
  8. Novel type I interferon IL-28A suppresses hepatitis C viral RNA replication. Virol J. 2005 Sep 07; 2:80.
    View in: PubMed
    Score: 0.005
  9. Gene expression associated with interferon alfa antiviral activity in an HCV replicon cell line. Hepatology. 2003 May; 37(5):1180-8.
    View in: PubMed
    Score: 0.004
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.