Connection

NANCY WEBB to Male

This is a "connection" page, showing publications NANCY WEBB has written about Male.
Connection Strength

0.164
  1. High-density lipoprotein inhibits serum amyloid A-mediated reactive oxygen species generation and NLRP3 inflammasome activation. J Biol Chem. 2018 08 24; 293(34):13257-13269.
    View in: PubMed
    Score: 0.024
  2. Group V secretory phospholipase A2 enhances the progression of angiotensin II-induced abdominal aortic aneurysms but confers protection against angiotensin II-induced cardiac fibrosis in apoE-deficient mice. Am J Pathol. 2012 Sep; 181(3):1088-98.
    View in: PubMed
    Score: 0.016
  3. Group X secretory phospholipase A2 negatively regulates ABCA1 and ABCG1 expression and cholesterol efflux in macrophages. Arterioscler Thromb Vasc Biol. 2010 Oct; 30(10):2014-21.
    View in: PubMed
    Score: 0.014
  4. Group X secretory phospholipase A2 negatively regulates adipogenesis in murine models. FASEB J. 2010 Nov; 24(11):4313-24.
    View in: PubMed
    Score: 0.014
  5. Group X secretory phospholipase A2 regulates the expression of steroidogenic acute regulatory protein (StAR) in mouse adrenal glands. J Biol Chem. 2010 Jun 25; 285(26):20031-9.
    View in: PubMed
    Score: 0.013
  6. The capacity of group V sPLA2 to increase atherogenicity of ApoE-/- and LDLR-/- mouse LDL in vitro predicts its atherogenic role in vivo. Arterioscler Thromb Vasc Biol. 2009 Apr; 29(4):532-8.
    View in: PubMed
    Score: 0.012
  7. Group V secretory phospholipase A2-modified low density lipoprotein promotes foam cell formation by a SR-A- and CD36-independent process that involves cellular proteoglycans. J Biol Chem. 2005 Sep 23; 280(38):32746-52.
    View in: PubMed
    Score: 0.010
  8. Antisense oligonucleotide targeting hepatic Serum Amyloid A limits the progression of angiotensin II-induced abdominal aortic aneurysm formation. Atherosclerosis. 2024 04; 391:117492.
    View in: PubMed
    Score: 0.009
  9. Macrophage-expressed group IIA secretory phospholipase A2 increases atherosclerotic lesion formation in LDL receptor-deficient mice. Arterioscler Thromb Vasc Biol. 2003 Feb 01; 23(2):263-8.
    View in: PubMed
    Score: 0.008
  10. Serum Amyloid A is not obligatory for high-fat, high-sucrose, cholesterol-fed diet-induced obesity and its metabolic and inflammatory complications. PLoS One. 2022; 17(4):e0266688.
    View in: PubMed
    Score: 0.008
  11. Adipocyte-Derived Serum Amyloid A Promotes Angiotensin II-Induced Abdominal Aortic Aneurysms in Obese C57BL/6J Mice. Arterioscler Thromb Vasc Biol. 2022 05; 42(5):632-643.
    View in: PubMed
    Score: 0.008
  12. Hepatocytes direct the formation of a pro-metastatic niche in the liver. Nature. 2019 03; 567(7747):249-252.
    View in: PubMed
    Score: 0.006
  13. Serum Amyloid A Is an Exchangeable Apolipoprotein. Arterioscler Thromb Vasc Biol. 2018 08; 38(8):1890-1900.
    View in: PubMed
    Score: 0.006
  14. Serum amyloid A3 is pro-atherogenic. Atherosclerosis. 2018 01; 268:32-35.
    View in: PubMed
    Score: 0.006
  15. A brief elevation of serum amyloid A is sufficient to increase atherosclerosis. J Lipid Res. 2015 Feb; 56(2):286-93.
    View in: PubMed
    Score: 0.005
  16. Deficiency of endogenous acute phase serum amyloid A does not affect atherosclerotic lesions in apolipoprotein E-deficient mice. Arterioscler Thromb Vasc Biol. 2014 Feb; 34(2):255-61.
    View in: PubMed
    Score: 0.004
  17. Serum amyloid A, but not C-reactive protein, stimulates vascular proteoglycan synthesis in a pro-atherogenic manner. Am J Pathol. 2008 Dec; 173(6):1902-10.
    View in: PubMed
    Score: 0.003
Connection Strength

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Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.