NK Cell Lectin-Like Receptor Subfamily K
"NK Cell Lectin-Like Receptor Subfamily K" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
An activating NK cell lectin-like receptor subfamily that regulates immune responses to INFECTION and NEOPLASMS. Members of this subfamily generally occur as homodimers.
Descriptor ID |
D055655
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MeSH Number(s) |
D12.776.543.750.705.895.800.910
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Concept/Terms |
NK Cell Lectin-Like Receptor Subfamily K- NK Cell Lectin-Like Receptor Subfamily K
- NK Cell Lectin Like Receptor Subfamily K
- Killer Cell Lectin-Like Receptor Subfamily K
- Killer Cell Lectin Like Receptor Subfamily K
NK Cell Lectin-Like Receptor Subfamily K, Member 1- NK Cell Lectin-Like Receptor Subfamily K, Member 1
- NK Cell Lectin Like Receptor Subfamily K, Member 1
- NKG2D Receptor
- Receptor, NKG2D
- CD314 Antigen
- Antigen, CD314
- NKG2-D Receptor
- NKG2 D Receptor
- Receptor, NKG2-D
- Killer Cell Lectin-Like Receptor Subfamily K, Member 1
- Killer Cell Lectin Like Receptor Subfamily K, Member 1
- NK Cell Receptor D
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Below are MeSH descriptors whose meaning is more general than "NK Cell Lectin-Like Receptor Subfamily K".
Below are MeSH descriptors whose meaning is more specific than "NK Cell Lectin-Like Receptor Subfamily K".
This graph shows the total number of publications written about "NK Cell Lectin-Like Receptor Subfamily K" by people in this website by year, and whether "NK Cell Lectin-Like Receptor Subfamily K" was a major or minor topic of these publications.
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Year | Major Topic | Minor Topic | Total |
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2005 | 0 | 2 | 2 |
2010 | 0 | 1 | 1 |
2014 | 2 | 0 | 2 |
2017 | 2 | 0 | 2 |
2018 | 2 | 0 | 2 |
2019 | 1 | 1 | 2 |
2022 | 0 | 1 | 1 |
2024 | 0 | 1 | 1 |
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Below are the most recent publications written about "NK Cell Lectin-Like Receptor Subfamily K" by people in Profiles.
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NK Receptor Signaling Lowers TCR Activation Threshold, Enhancing Selective Recognition of Cancer Cells by TAA-Specific CTLs. Cancer Immunol Res. 2024 Oct 01; 12(10):1421-1437.
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Enhancing cancer immunotherapy with Anti-NKG2D/IL-15(N72D)/Sushi fusion protein: Targeting cytotoxic immune cells and boosting IL-15 efficacy. Cytokine. 2024 04; 176:156505.
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STAT3 gain-of-function mutations connect leukemia with autoimmune disease by pathological NKG2Dhi CD8+?T?cell dysregulation and accumulation. Immunity. 2022 12 13; 55(12):2386-2404.e8.
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NK Cells Expressing a Chimeric Activating Receptor Eliminate MDSCs and Rescue Impaired CAR-T Cell Activity against Solid Tumors. Cancer Immunol Res. 2019 03; 7(3):363-375.
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Differential role of natural killer group 2D in recognition and cytotoxicity of hepatocyte-like cells derived from embryonic stem cells and induced pluripotent stem cells. Am J Transplant. 2019 06; 19(6):1652-1662.
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High-throughput T cell receptor sequencing identifies clonally expanded CD8+ T cell populations in alopecia areata. JCI Insight. 2018 10 04; 3(19).
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A Valine Mismatch at Position 129 of MICA Is an Independent Predictor of Cytomegalovirus Infection and Acute Kidney Rejection in Simultaneous Pancreas?Kidney Transplantation Recipients. Int J Mol Sci. 2018 Sep 04; 19(9).
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The Donor Major Histocompatibility Complex Class I Chain-Related Molecule A Allele rs2596538 G Predicts Cytomegalovirus Viremia in Kidney Transplant Recipients. Front Immunol. 2018; 9:917.
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MICA-Expressing Monocytes Enhance Natural Killer Cell Fc Receptor-Mediated Antitumor Functions. Cancer Immunol Res. 2017 09; 5(9):778-789.
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The histone deacetylase inhibitor valproic acid inhibits NKG2D expression in natural killer cells through suppression of STAT3 and HDAC3. Sci Rep. 2017 03 24; 7:45266.