Co-Authors
This is a "connection" page, showing publications co-authored by STEVEN HSESHENG LIN and NHAT TU LE.
Connection Strength
1.281
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Premature senescence and cardiovascular disease following cancer treatments: mechanistic insights. Front Cardiovasc Med. 2023; 10:1212174.
Score: 0.230
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Endothelial activation and fibrotic changes are impeded by laminar flow-induced CHK1-SENP2 activity through mechanisms distinct from endothelial-to-mesenchymal cell transition. Front Cardiovasc Med. 2023; 10:1187490.
Score: 0.230
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Possible molecular mechanisms underlying the development of atherosclerosis in cancer survivors. Front Cardiovasc Med. 2023; 10:1186679.
Score: 0.226
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Corrigendum: p90RSK-MAGI1 Module Controls Endothelial Permeability by Post-translational Modifications of MAGI1 and Hippo Pathway. Front Cardiovasc Med. 2021; 8:663486.
Score: 0.193
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p90RSK-MAGI1 Module Controls Endothelial Permeability by Post-translational Modifications of MAGI1 and Hippo Pathway. Front Cardiovasc Med. 2020; 7:542485.
Score: 0.189
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An ERK5-NRF2 Axis Mediates Senescence-Associated Stemness and Atherosclerosis. Circ Res. 2023 06 23; 133(1):25-44.
Score: 0.056
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Radiation therapy induces immunosenescence mediated by p90RSK. Front Cardiovasc Med. 2022; 9:988713.
Score: 0.054
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Cancer treatment-induced NAD+ depletion in premature senescence and late cardiovascular complications. J Cardiovasc Aging. 2022; 2.
Score: 0.052
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Nucleus-mitochondria positive feedback loop formed by ERK5 S496 phosphorylation-mediated poly (ADP-ribose) polymerase activation provokes persistent pro-inflammatory senescent phenotype and accelerates coronary atherosclerosis after chemo-radiation. Redox Biol. 2021 11; 47:102132.
Score: 0.050