"Rats, Inbred SHR" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A strain of Rattus norvegicus with elevated blood pressure used as a model for studying hypertension and stroke.
| Descriptor ID |
D011918
|
| MeSH Number(s) |
B01.050.050.199.520.760.300 B01.050.150.900.649.313.992.635.505.700.400.300
|
| Concept/Terms |
Rats, Inbred SHR- Rats, Inbred SHR
- Inbred SHR Rat
- Inbred SHR Rats
- Rat, Inbred SHR
- SHR Rat, Inbred
- SHR Rats, Inbred
- Rats, SHR
- Rat, SHR
- SHR Rat
- SHR Rats
- Rats, Spontaneously Hypertensive
- Rat, Spontaneously Hypertensive
- Spontaneously Hypertensive Rat
- Spontaneously Hypertensive Rats
|
Below are MeSH descriptors whose meaning is more general than "Rats, Inbred SHR".
Below are MeSH descriptors whose meaning is more specific than "Rats, Inbred SHR".
This graph shows the total number of publications written about "Rats, Inbred SHR" by people in this website by year, and whether "Rats, Inbred SHR" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1994 | 1 | 0 | 1 |
| 1995 | 0 | 2 | 2 |
| 1997 | 0 | 1 | 1 |
| 1998 | 0 | 1 | 1 |
| 2001 | 0 | 2 | 2 |
| 2002 | 0 | 1 | 1 |
| 2003 | 0 | 3 | 3 |
| 2004 | 0 | 1 | 1 |
| 2005 | 1 | 0 | 1 |
| 2006 | 1 | 2 | 3 |
| 2007 | 0 | 2 | 2 |
| 2008 | 0 | 3 | 3 |
| 2009 | 0 | 2 | 2 |
| 2010 | 0 | 3 | 3 |
| 2011 | 1 | 4 | 5 |
| 2012 | 0 | 3 | 3 |
| 2013 | 0 | 4 | 4 |
| 2014 | 0 | 5 | 5 |
| 2015 | 0 | 4 | 4 |
| 2016 | 0 | 3 | 3 |
| 2017 | 0 | 1 | 1 |
| 2018 | 0 | 2 | 2 |
| 2019 | 0 | 2 | 2 |
| 2020 | 0 | 3 | 3 |
| 2021 | 0 | 3 | 3 |
| 2022 | 0 | 2 | 2 |
| 2023 | 0 | 1 | 1 |
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Below are the most recent publications written about "Rats, Inbred SHR" by people in Profiles.
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DNA demethylation in the hypothalamus promotes transcription of Agtr1a and Slc12a2 and hypertension development. J Biol Chem. 2024 Feb; 300(2):105597.
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Alterations of the gut microbial community structure and function with aging in the spontaneously hypertensive stroke prone rat. Sci Rep. 2022 05 20; 12(1):8534.
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a2d-1 protein promotes synaptic expression of Ca2+ permeable-AMPA receptors by inhibiting GluA1/GluA2 heteromeric assembly in the hypothalamus in hypertension. J Neurochem. 2022 04; 161(1):40-52.
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a2d-1-Dependent NMDA Receptor Activity in the Hypothalamus Is an Effector of Genetic-Environment Interactions That Drive Persistent Hypertension. J Neurosci. 2021 07 28; 41(30):6551-6563.
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Restructuring the Gut Microbiota by Intermittent Fasting Lowers Blood Pressure. Circ Res. 2021 04 30; 128(9):1240-1254.
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The gut microbiome contributes to blood-brain barrier disruption in spontaneously hypertensive stroke prone rats. FASEB J. 2021 02; 35(2):e21201.
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Group III metabotropic glutamate receptors regulate hypothalamic presympathetic neurons through opposing presynaptic and postsynaptic actions in hypertension. Neuropharmacology. 2020 09 01; 174:108159.
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Natural genetic variation in Stim1 creates stroke in the spontaneously hypertensive rat. Genes Immun. 2020 05; 21(3):182-192.
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Metformin Improves Cardiac Metabolism and Function, and Prevents Left Ventricular Hypertrophy in Spontaneously Hypertensive Rats. J Am Heart Assoc. 2020 04 07; 9(7):e015154.
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Stim1 Polymorphism Disrupts Immune Signaling and Creates Renal Injury in Hypertension. J Am Heart Assoc. 2020 03 03; 9(5):e014142.