"Phentolamine" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A nonselective alpha-adrenergic antagonist. It is used in the treatment of hypertension and hypertensive emergencies, pheochromocytoma, vasospasm of RAYNAUD DISEASE and frostbite, clonidine withdrawal syndrome, impotence, and peripheral vascular disease.
| Descriptor ID |
D010646
|
| MeSH Number(s) |
D03.383.129.308.754
|
| Concept/Terms |
Phentolamine Mesylate- Phentolamine Mesylate
- Mesylate, Phentolamine
- Phentolamine Methanesulfonate
- Methanesulfonate, Phentolamine
- Phentolamine Mesilate
- Mesilate, Phentolamine
|
Below are MeSH descriptors whose meaning is more general than "Phentolamine".
Below are MeSH descriptors whose meaning is more specific than "Phentolamine".
This graph shows the total number of publications written about "Phentolamine" by people in this website by year, and whether "Phentolamine" was a major or minor topic of these publications.
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click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2007 | 0 | 1 | 1 |
| 2021 | 0 | 1 | 1 |
| 2023 | 0 | 1 | 1 |
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click here.
Below are the most recent publications written about "Phentolamine" by people in Profiles.
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Intravenous aviptadil and remdesivir for treatment of COVID-19-associated hypoxaemic respiratory failure in the USA (TESICO): a randomised, placebo-controlled trial. Lancet Respir Med. 2023 09; 11(9):791-803.
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Alpha-blockade during congenital heart surgery admissions: analysis from national database. Cardiol Young. 2022 Jul; 32(7):1136-1142.
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GRK2 in sensory neurons regulates epinephrine-induced signalling and duration of mechanical hyperalgesia. Pain. 2011 Jul; 152(7):1649-1658.
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Electroacupuncture suppresses capsaicin-induced secondary hyperalgesia through an endogenous spinal opioid mechanism. Pain. 2009 Oct; 145(3):332-340.
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Extracellular signal-regulated kinase 1/2-mediated transcriptional regulation of G-protein-coupled receptor kinase 3 expression in neuronal cells. J Pharmacol Exp Ther. 2007 Apr; 321(1):51-9.
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Acidosis-induced glucose intolerance is not prevented by adrenergic blockade. Am J Physiol. 1988 Dec; 255(6 Pt 1):E812-23.
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Epinephrine and norepinephrine are cleared through beta-adrenergic, but not alpha-adrenergic, mechanisms in man. Metabolism. 1980 Nov; 29(11 Suppl 1):1114-8.
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Adrenergic mechanisms of catecholamine action on glucose homeostasis in man. Metabolism. 1980 Nov; 29(11 Suppl 1):1155-63.
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Effect of alpha-adrenergic stimulation and its blockade on glucose turnover in man. Am J Physiol. 1980 May; 238(5):E467-72.
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Adrenergic mechanisms for the effects of epinephrine on glucose production and clearance in man. J Clin Invest. 1980 Mar; 65(3):682-9.