Connection

INDIRA MYSOREKAR to Escherichia coli Infections

This is a "connection" page, showing publications INDIRA MYSOREKAR has written about Escherichia coli Infections.
Connection Strength

6.142
  1. Supraphysiologic Vaginal Estrogen Therapy in Aged Mice Mitigates Age-Associated Bladder Inflammatory Response to Urinary Tract Infections. Urogynecology (Phila). 2023 04 01; 29(4):430-442.
    View in: PubMed
    Score: 0.643
  2. Gut-bladder axis enters the stage: Implication for recurrent urinary tract infections. Cell Host Microbe. 2022 08 10; 30(8):1066-1069.
    View in: PubMed
    Score: 0.634
  3. PITing it forward: A new link in the journey of uropathogenic E.?coli in the urothelium. Cell Rep. 2022 04 26; 39(4):110758.
    View in: PubMed
    Score: 0.621
  4. NRF2 promotes urothelial cell response to bacterial infection by regulating reactive oxygen species and RAB27B expression. Cell Rep. 2021 10 19; 37(3):109856.
    View in: PubMed
    Score: 0.599
  5. Dietary restriction of iron availability attenuates UPEC pathogenesis in a mouse model of urinary tract infection. Am J Physiol Renal Physiol. 2019 05 01; 316(5):F814-F822.
    View in: PubMed
    Score: 0.497
  6. A non-canonical autophagy-dependent role of the ATG16L1T300A variant in urothelial vesicular trafficking and uropathogenic Escherichia coli persistence. Autophagy. 2019 03; 15(3):527-542.
    View in: PubMed
    Score: 0.489
  7. Trans-mission control in the urinary tract: Local cytokine regulation of monocyte proliferation to combat infection. J Leukoc Biol. 2018 01; 103(1):5-7.
    View in: PubMed
    Score: 0.459
  8. Macrophagic control of the response to uropathogenic E. coli infection by regulation of iron retention in an IL-6-dependent manner. Immun Inflamm Dis. 2016 12; 4(4):413-426.
    View in: PubMed
    Score: 0.420
  9. Estrogenic modulation of uropathogenic Escherichia coli infection pathogenesis in a murine menopause model. Infect Immun. 2013 Mar; 81(3):733-9.
    View in: PubMed
    Score: 0.325
  10. Atg16L1 deficiency confers protection from uropathogenic Escherichia coli infection in vivo. Proc Natl Acad Sci U S A. 2012 Jul 03; 109(27):11008-13.
    View in: PubMed
    Score: 0.314
  11. Bone morphogenetic protein 4 signaling regulates epithelial renewal in the urinary tract in response to uropathogenic infection. Cell Host Microbe. 2009 May 08; 5(5):463-75.
    View in: PubMed
    Score: 0.253
  12. Mechanisms of uropathogenic Escherichia coli persistence and eradication from the urinary tract. Proc Natl Acad Sci U S A. 2006 Sep 19; 103(38):14170-5.
    View in: PubMed
    Score: 0.210
  13. Bacteriophage-mediated reduction of uropathogenic E. coli from the urogenital epithelium. Infect Immun. 2026 Apr 15; 94(4):e0054325.
    View in: PubMed
    Score: 0.203
  14. A broad-spectrum antibiotic, DCAP, reduces uropathogenic Escherichia coli infection and enhances vorinostat anticancer activity by modulating autophagy. Cell Death Dis. 2018 07 13; 9(7):780.
    View in: PubMed
    Score: 0.119
  15. Ferritinophagy drives uropathogenic Escherichia coli persistence in bladder epithelial cells. Autophagy. 2016 05 03; 12(5):850-63.
    View in: PubMed
    Score: 0.103
  16. Estrogen affects the glycosaminoglycan layer of the murine bladder. Female Pelvic Med Reconstr Surg. 2012 May-Jun; 18(3):148-52.
    View in: PubMed
    Score: 0.078
  17. Early severe inflammatory responses to uropathogenic E. coli predispose to chronic and recurrent urinary tract infection. PLoS Pathog. 2010 Aug 12; 6(8):e1001042.
    View in: PubMed
    Score: 0.069
  18. LeuX tRNA-dependent and -independent mechanisms of Escherichia coli pathogenesis in acute cystitis. Mol Microbiol. 2008 Jan; 67(1):116-28.
    View in: PubMed
    Score: 0.057
  19. Urinary tract infections in children. Nat Rev Urol. 2026 Sep; 23(9):592-606.
    View in: PubMed
    Score: 0.051
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.