SUMIMASA ARIMURA to Mice
This is a "connection" page, showing publications SUMIMASA ARIMURA has written about Mice.
Connection Strength
0.166
-
Loss of Dok-3 in Non-tumor Cells Induces Malignant Transformation of Benign Epithelial Tumor Cells of the Intestine. Cancer Res Commun. 2022 12; 2(12):1590-1600.
Score: 0.059
-
Neuromuscular disease. DOK7 gene therapy benefits mouse models of diseases characterized by defects in the neuromuscular junction. Science. 2014 Sep 19; 345(6203):1505-8.
Score: 0.033
-
Reduced level of smoothened suppresses intestinal tumorigenesis by down-regulation of Wnt signaling. Gastroenterology. 2009 Aug; 137(2):629-38.
Score: 0.023
-
Metabolic regulator ERR? governs gastric stem cell differentiation into acid-secreting parietal cells. Cell Stem Cell. 2024 Jun 06; 31(6):886-903.e8.
Score: 0.016
-
Functional characterization of age-dependent p16 epimutation reveals biological drivers and therapeutic targets for colorectal cancer. J Exp Clin Cancer Res. 2023 May 04; 42(1):113.
Score: 0.015
-
Dok-3 and Dok-1/-2 adaptors play distinctive roles in cell fusion and proliferation during osteoclastogenesis and cooperatively protect mice from osteopenia. Biochem Biophys Res Commun. 2018 04 15; 498(4):967-974.
Score: 0.011
-
Loss of Dok-1 and Dok-2 in mice causes severe experimental colitis accompanied by reduced expression of IL-17A and IL-22. Biochem Biophys Res Commun. 2016 09 09; 478(1):135-142.
Score: 0.009