Connection

HAMED JAFAR-NEJAD to Animals

This is a "connection" page, showing publications HAMED JAFAR-NEJAD has written about Animals.
Connection Strength

0.967
  1. Gut barrier defects, intestinal immune hyperactivation and enhanced lipid catabolism drive lethality in NGLY1-deficient Drosophila. Nat Commun. 2023 09 13; 14(1):5667.
    View in: PubMed
    Score: 0.048
  2. ASO silencing of a glycosyltransferase, Poglut1 , improves the liver phenotypes in mouse models of Alagille syndrome. Hepatology. 2023 11 01; 78(5):1337-1351.
    View in: PubMed
    Score: 0.047
  3. NGLY1 Deficiency, a Congenital Disorder of Deglycosylation: From Disease Gene Function to Pathophysiology. Cells. 2022 03 29; 11(7).
    View in: PubMed
    Score: 0.044
  4. Tracing the NGLY1 footprints: insights from Drosophila. J Biochem. 2022 Feb 21; 171(2):153-160.
    View in: PubMed
    Score: 0.043
  5. Multifaceted regulation of Notch signaling by glycosylation. Glycobiology. 2021 01 09; 31(1):8-28.
    View in: PubMed
    Score: 0.040
  6. A conserved role for AMP-activated protein kinase in NGLY1 deficiency. PLoS Genet. 2020 12; 16(12):e1009258.
    View in: PubMed
    Score: 0.040
  7. Regulation of BMP4/Dpp retrotranslocation and signaling by deglycosylation. Elife. 2020 07 28; 9.
    View in: PubMed
    Score: 0.039
  8. Sox9 Is a Modifier of the Liver Disease Severity in a Mouse Model of Alagille Syndrome. Hepatology. 2020 04; 71(4):1331-1349.
    View in: PubMed
    Score: 0.038
  9. Glycosylation of Specific Notch EGF Repeats by O-Fut1 and Fringe Regulates Notch Signaling in Drosophila. Cell Rep. 2019 11 12; 29(7):2054-2066.e6.
    View in: PubMed
    Score: 0.037
  10. The Roles of Notch Signaling in Liver Development and Disease. Biomolecules. 2019 10 14; 9(10).
    View in: PubMed
    Score: 0.037
  11. Determining Bile Duct Density in the Mouse Liver. J Vis Exp. 2019 04 30; (146).
    View in: PubMed
    Score: 0.036
  12. Sensitized genetic backgrounds reveal differential roles for EGF repeat xylosyltransferases in Drosophila Notch signaling. Glycobiology. 2018 11 01; 28(11):849-859.
    View in: PubMed
    Score: 0.035
  13. A New Model of Alagille Syndrome With Broad Phenotypic Representation. Gastroenterology. 2018 03; 154(4):803-806.
    View in: PubMed
    Score: 0.033
  14. Cell Aggregation Assays to Evaluate the Binding of the Drosophila Notch with Trans-Ligands and its Inhibition by Cis-Ligands. J Vis Exp. 2018 01 02; (131).
    View in: PubMed
    Score: 0.033
  15. Tissue-specific regulation of BMP signaling by Drosophila N-glycanase 1. Elife. 2017 08 04; 6.
    View in: PubMed
    Score: 0.032
  16. Xylosylation of the Notch receptor preserves the balance between its activation by trans-Delta and inhibition by cis-ligands in Drosophila. PLoS Genet. 2017 04; 13(4):e1006723.
    View in: PubMed
    Score: 0.031
  17. Jagged1 heterozygosity in mice results in a congenital cholangiopathy which is reversed by concomitant deletion of one copy of Poglut1 (Rumi). Hepatology. 2016 Feb; 63(2):550-65.
    View in: PubMed
    Score: 0.028
  18. The multiple roles of epidermal growth factor repeat O-glycans in animal development. Glycobiology. 2015 Oct; 25(10):1027-42.
    View in: PubMed
    Score: 0.028
  19. The protein O-glucosyltransferase Rumi modifies eyes shut to promote rhabdomere separation in Drosophila. PLoS Genet. 2014 Nov; 10(11):e1004795.
    View in: PubMed
    Score: 0.026
  20. Structure-function analysis of Drosophila Notch using genomic rescue transgenes. Methods Mol Biol. 2014; 1187:29-46.
    View in: PubMed
    Score: 0.025
  21. Negative regulation of notch signaling by xylose. PLoS Genet. 2013 Jun; 9(6):e1003547.
    View in: PubMed
    Score: 0.024
  22. Multiple O-glucosylation sites on Notch function as a buffer against temperature-dependent loss of signaling. Development. 2011 Aug; 138(16):3569-78.
    View in: PubMed
    Score: 0.021
  23. Regulation of mammalian Notch signaling and embryonic development by the protein O-glucosyltransferase Rumi. Development. 2011 May; 138(10):1925-34.
    View in: PubMed
    Score: 0.020
  24. Role of glycans and glycosyltransferases in the regulation of Notch signaling. Glycobiology. 2010 Aug; 20(8):931-49.
    View in: PubMed
    Score: 0.019
  25. Regulation of notch signaling via O-glucosylation insights from Drosophila studies. Methods Enzymol. 2010; 480:375-98.
    View in: PubMed
    Score: 0.019
  26. Drosophila embryonic 'fibroblasts': extending mutant analysis in vitro. Fly (Austin). 2008 Nov-Dec; 2(6):306-9.
    View in: PubMed
    Score: 0.017
  27. Senseless and Daughterless confer neuronal identity to epithelial cells in the Drosophila wing margin. Development. 2006 May; 133(9):1683-92.
    View in: PubMed
    Score: 0.014
  28. Sec15, a component of the exocyst, promotes notch signaling during the asymmetric division of Drosophila sensory organ precursors. Dev Cell. 2005 Sep; 9(3):351-63.
    View in: PubMed
    Score: 0.014
  29. Gfi/Pag-3/senseless zinc finger proteins: a unifying theme? Mol Cell Biol. 2004 Oct; 24(20):8803-12.
    View in: PubMed
    Score: 0.013
  30. Senseless acts as a binary switch during sensory organ precursor selection. Genes Dev. 2003 Dec 01; 17(23):2966-78.
    View in: PubMed
    Score: 0.012
  31. Numb: "Adapting" notch for endocytosis. Dev Cell. 2002 Aug; 3(2):155-6.
    View in: PubMed
    Score: 0.011
  32. POGLUT1 biallelic mutations cause myopathy with reduced satellite cells, a-dystroglycan hypoglycosylation and a distinctive radiological pattern. Acta Neuropathol. 2020 03; 139(3):565-582.
    View in: PubMed
    Score: 0.009
  33. Mapping Sites of O-Glycosylation and Fringe Elongation on Drosophila Notch. J Biol Chem. 2016 07 29; 291(31):16348-60.
    View in: PubMed
    Score: 0.007
  34. Fringe proteins modulate Notch-ligand cis and trans interactions to specify signaling states. Elife. 2014 Sep 25; 3:e02950.
    View in: PubMed
    Score: 0.007
  35. Rumi functions as both a protein O-glucosyltransferase and a protein O-xylosyltransferase. Proc Natl Acad Sci U S A. 2011 Oct 04; 108(40):16600-5.
    View in: PubMed
    Score: 0.005
  36. Novel function of the class I bHLH protein Daughterless in the negative regulation of proneural gene expression in the Drosophila eye. EMBO Rep. 2008 Nov; 9(11):1128-33.
    View in: PubMed
    Score: 0.004
  37. Rumi is a CAP10 domain glycosyltransferase that modifies Notch and is required for Notch signaling. Cell. 2008 Jan 25; 132(2):247-58.
    View in: PubMed
    Score: 0.004
  38. Differential repression by freud-1/CC2D1A at a polymorphic site in the dopamine-D2 receptor gene. J Biol Chem. 2007 Jul 20; 282(29):20897-905.
    View in: PubMed
    Score: 0.004
  39. Senseless physically interacts with proneural proteins and functions as a transcriptional co-activator. Development. 2006 May; 133(10):1979-89.
    View in: PubMed
    Score: 0.004
  40. The tumour-suppressor genes NF2/Merlin and Expanded act through Hippo signalling to regulate cell proliferation and apoptosis. Nat Cell Biol. 2006 Jan; 8(1):27-36.
    View in: PubMed
    Score: 0.004
  41. The AXH domain of Ataxin-1 mediates neurodegeneration through its interaction with Gfi-1/Senseless proteins. Cell. 2005 Aug 26; 122(4):633-44.
    View in: PubMed
    Score: 0.003
  42. Evolution of neural precursor selection: functional divergence of proneural proteins. Development. 2004 Apr; 131(8):1679-89.
    View in: PubMed
    Score: 0.003
  43. Mapping Drosophila mutations with molecularly defined P element insertions. Proc Natl Acad Sci U S A. 2003 Sep 16; 100(19):10860-5.
    View in: PubMed
    Score: 0.003
  44. Freud-1: A neuronal calcium-regulated repressor of the 5-HT1A receptor gene. J Neurosci. 2003 Aug 13; 23(19):7415-25.
    View in: PubMed
    Score: 0.003
  45. Novel dual repressor elements for neuronal cell-specific transcription of the rat 5-HT1A receptor gene. J Biol Chem. 2000 Mar 17; 275(11):8161-8.
    View in: PubMed
    Score: 0.002
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.