Connection

JEFFREY NOEBELS to Epilepsy, Absence

This is a "connection" page, showing publications JEFFREY NOEBELS has written about Epilepsy, Absence.
Connection Strength

4.482
  1. Adult loss of Cacna1a in mice recapitulates childhood absence epilepsy by distinct thalamic bursting mechanisms. Brain. 2020 01 01; 143(1):161-174.
    View in: PubMed
    Score: 0.663
  2. Persistent aberrant cortical phase-amplitude coupling following seizure treatment in absence epilepsy models. J Physiol. 2017 12 01; 595(23):7249-7260.
    View in: PubMed
    Score: 0.566
  3. Isolated P/Q Calcium Channel Deletion in Layer VI Corticothalamic Neurons Generates Absence Epilepsy. J Neurosci. 2016 Jan 13; 36(2):405-18.
    View in: PubMed
    Score: 0.504
  4. Shift in interictal relative gamma power as a novel biomarker for drug response in two mouse models of absence epilepsy. Epilepsia. 2016 Jan; 57(1):79-88.
    View in: PubMed
    Score: 0.501
  5. Monogenic models of absence epilepsy: windows into the complex balance between inhibition and excitation in thalamocortical microcircuits. Prog Brain Res. 2014; 213:223-52.
    View in: PubMed
    Score: 0.438
  6. Expanded alternative splice isoform profiling of the mouse Cav3.1/alpha1G T-type calcium channel. BMC Mol Biol. 2009 May 29; 10:53.
    View in: PubMed
    Score: 0.318
  7. Genetic enhancement of thalamocortical network activity by elevating alpha 1g-mediated low-voltage-activated calcium current induces pure absence epilepsy. J Neurosci. 2009 Feb 11; 29(6):1615-25.
    View in: PubMed
    Score: 0.312
  8. Elevated thalamic low-voltage-activated currents precede the onset of absence epilepsy in the SNAP25-deficient mouse mutant coloboma. J Neurosci. 2004 Jun 02; 24(22):5239-48.
    View in: PubMed
    Score: 0.225
  9. Mutations in high-voltage-activated calcium channel genes stimulate low-voltage-activated currents in mouse thalamic relay neurons. J Neurosci. 2002 Aug 01; 22(15):6362-71.
    View in: PubMed
    Score: 0.198
  10. Single gene defects in mice: the role of voltage-dependent calcium channels in absence models. Epilepsy Res. 1999 Sep; 36(2-3):111-22.
    View in: PubMed
    Score: 0.162
  11. Asynchronous suppression of visual cortex during absence seizures in stargazer mice. Nat Commun. 2018 05 16; 9(1):1938.
    View in: PubMed
    Score: 0.148
  12. Increased excitability and inward rectification in layer V cortical pyramidal neurons in the epileptic mutant mouse Stargazer. J Neurophysiol. 1997 Feb; 77(2):621-31.
    View in: PubMed
    Score: 0.135
  13. Single locus mutations in mice expressing generalized spike-wave absence epilepsies. Ital J Neurol Sci. 1995 Feb-Mar; 16(1-2):107-11.
    View in: PubMed
    Score: 0.118
  14. Postnatal loss of P/Q-type channels confined to rhombic-lip-derived neurons alters synaptic transmission at the parallel fiber to purkinje cell synapse and replicates genomic Cacna1a mutation phenotype of ataxia and seizures in mice. J Neurosci. 2013 Mar 20; 33(12):5162-74.
    View in: PubMed
    Score: 0.104
  15. Delayed postnatal loss of P/Q-type calcium channels recapitulates the absence epilepsy, dyskinesia, and ataxia phenotypes of genomic Cacna1a mutations. J Neurosci. 2011 Mar 16; 31(11):4311-26.
    View in: PubMed
    Score: 0.090
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.