Connection

GRAEME MARDON to Leber Congenital Amaurosis

This is a "connection" page, showing publications GRAEME MARDON has written about Leber Congenital Amaurosis.
Connection Strength

1.463
  1. AAV8(Y733F)-mediated gene therapy in a Spata7 knockout mouse model of Leber congenital amaurosis and retinitis pigmentosa. Gene Ther. 2015 Aug; 22(8):619-27.
    View in: PubMed
    Score: 0.476
  2. CRISPR-engineered mosaicism rapidly reveals that loss of Kcnj13 function in mice mimics human disease phenotypes. Sci Rep. 2015 Feb 10; 5:8366.
    View in: PubMed
    Score: 0.468
  3. NMNAT1 E257K variant, associated with Leber Congenital Amaurosis (LCA9), causes a mild retinal degeneration phenotype. Exp Eye Res. 2018 08; 173:32-43.
    View in: PubMed
    Score: 0.146
  4. Mutations in NMNAT1 cause Leber congenital amaurosis and identify a new disease pathway for retinal degeneration. Nat Genet. 2012 Sep; 44(9):1035-9.
    View in: PubMed
    Score: 0.098
  5. Exome capture sequencing identifies a novel mutation in BBS4. Mol Vis. 2011; 17:3529-40.
    View in: PubMed
    Score: 0.094
  6. Whole-exome sequencing identifies ALMS1, IQCB1, CNGA3, and MYO7A mutations in patients with Leber congenital amaurosis. Hum Mutat. 2011 Dec; 32(12):1450-9.
    View in: PubMed
    Score: 0.093
  7. Potential involvement of more than one locus in trait manifestation for individuals with Leber congenital amaurosis. Hum Genet. 2011 Mar; 129(3):319-27.
    View in: PubMed
    Score: 0.088
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.