Connection

DAVID SHEIKH-HAMAD to Glycoproteins

This is a "connection" page, showing publications DAVID SHEIKH-HAMAD has written about Glycoproteins.
Connection Strength

4.266
  1. Stanniocalcin-1 inhibits thrombin-induced signaling and protects from bleomycin-induced lung injury. Sci Rep. 2015 Dec 07; 5:18117.
    View in: PubMed
    Score: 0.410
  2. Severe Nephrotoxic Nephritis following Conditional and Kidney-Specific Knockdown of Stanniocalcin-1. PLoS One. 2015; 10(9):e0138440.
    View in: PubMed
    Score: 0.404
  3. Stanniocalcin-1 inhibits renal ischemia/reperfusion injury via an AMP-activated protein kinase-dependent pathway. J Am Soc Nephrol. 2015 Feb; 26(2):364-78.
    View in: PubMed
    Score: 0.371
  4. AKI after conditional and kidney-specific knockdown of stanniocalcin-1. J Am Soc Nephrol. 2014 Oct; 25(10):2303-15.
    View in: PubMed
    Score: 0.365
  5. Overexpression of stanniocalcin-1 inhibits reactive oxygen species and renal ischemia/reperfusion injury in mice. Kidney Int. 2012 Oct; 82(8):867-77.
    View in: PubMed
    Score: 0.322
  6. Human stanniocalcin-1 suppresses angiotensin II-induced superoxide generation in cardiomyocytes through UCP3-mediated anti-oxidant pathway. PLoS One. 2012; 7(5):e36994.
    View in: PubMed
    Score: 0.321
  7. Mammalian stanniocalcin-1 activates mitochondrial antioxidant pathways: new paradigms for regulation of macrophages and endothelium. Am J Physiol Renal Physiol. 2010 Feb; 298(2):F248-54.
    View in: PubMed
    Score: 0.264
  8. Stanniocalcin-1 suppresses superoxide generation in macrophages through induction of mitochondrial UCP2. J Leukoc Biol. 2009 Oct; 86(4):981-8.
    View in: PubMed
    Score: 0.263
  9. Anti-inflammatory and renal protective actions of stanniocalcin-1 in a model of anti-glomerular basement membrane glomerulonephritis. Am J Pathol. 2009 Apr; 174(4):1368-78.
    View in: PubMed
    Score: 0.256
  10. Stanniocalcin-1 regulates endothelial gene expression and modulates transendothelial migration of leukocytes. Am J Physiol Renal Physiol. 2007 Feb; 292(2):F895-904.
    View in: PubMed
    Score: 0.217
  11. Stanniocalcin-1, an inhibitor of macrophage chemotaxis and chemokinesis. Am J Physiol Renal Physiol. 2004 Feb; 286(2):F356-62.
    View in: PubMed
    Score: 0.177
  12. Stanniocalcin-1 is a naturally occurring L-channel inhibitor in cardiomyocytes: relevance to human heart failure. Am J Physiol Heart Circ Physiol. 2003 Jul; 285(1):H442-8.
    View in: PubMed
    Score: 0.170
  13. Interactions between leucines within the signal peptides of megalin and stanniocalcin-1 are crucial for regulation of mitochondrial metabolism. Lab Invest. 2022 05; 102(5):534-544.
    View in: PubMed
    Score: 0.156
  14. Regulation of stanniocalcin in MDCK cells by hypertonicity and extracellular calcium. Am J Physiol Renal Physiol. 2000 Mar; 278(3):F417-24.
    View in: PubMed
    Score: 0.137
  15. Stanniocalcin-1 is a Modifier of Oxygen-Induced Retinopathy Severity. Curr Eye Res. 2020 01; 45(1):46-51.
    View in: PubMed
    Score: 0.133
  16. Megalin mediates plasma membrane to mitochondria cross-talk and regulates mitochondrial metabolism. Cell Mol Life Sci. 2018 Nov; 75(21):4021-4040.
    View in: PubMed
    Score: 0.122
  17. Stanniocalcin-1 Is an Ocular Hypotensive Agent and a Downstream Effector Molecule That Is Necessary for the Intraocular Pressure-Lowering Effects of Latanoprost. Invest Ophthalmol Vis Sci. 2017 05 01; 58(5):2715-2724.
    View in: PubMed
    Score: 0.113
  18. Human stanniocalcin-1 blocks TNF-alpha-induced monolayer permeability in human coronary artery endothelial cells. Arterioscler Thromb Vasc Biol. 2008 May; 28(5):906-12.
    View in: PubMed
    Score: 0.060
  19. Cell apoptosis and proliferation in obstructive uropathy. Semin Nephrol. 1998 Nov; 18(6):641-51.
    View in: PubMed
    Score: 0.008
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.