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NIKOLAOS FRANGOGIANNIS to Mice, Knockout

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This is a "connection" page, showing publications NIKOLAOS FRANGOGIANNIS has written about Mice, Knockout.
NIKOLAOS FRANGOGIANNIS
Connection Strength
1.783
Mice, Knockout
  1. Protective effects of macrophage-specific integrin a5 in myocardial infarction are associated with accentuated angiogenesis. Nat Commun. 2023 Nov 20; 14(1):7555.
    View in: PubMed
    Score: 0.118
  2. Cardiac Pericytes Acquire a Fibrogenic Phenotype and Contribute to Vascular Maturation After Myocardial Infarction. Circulation. 2023 09 12; 148(11):882-898.
    View in: PubMed
    Score: 0.115
  3. Differential effects of Smad2 and Smad3 in regulation of macrophage phenotype and function in the infarcted myocardium. J Mol Cell Cardiol. 2022 10; 171:1-15.
    View in: PubMed
    Score: 0.107
  4. The role of Smad2 and Smad3 in regulating homeostatic functions of fibroblasts in vitro and in adult mice. Biochim Biophys Acta Mol Cell Res. 2020 07; 1867(7):118703.
    View in: PubMed
    Score: 0.091
  5. Macrophage Smad3 Protects the Infarcted Heart, Stimulating Phagocytosis and Regulating Inflammation. Circ Res. 2019 06 21; 125(1):55-70.
    View in: PubMed
    Score: 0.086
  6. Distinct roles of myofibroblast-specific Smad2 and Smad3 signaling in repair and remodeling of the infarcted heart. J Mol Cell Cardiol. 2019 07; 132:84-97.
    View in: PubMed
    Score: 0.086
  7. Protective Effects of Activated Myofibroblasts in the Pressure-Overloaded Myocardium Are Mediated Through Smad-Dependent Activation of a Matrix-Preserving Program. Circ Res. 2019 04 12; 124(8):1214-1227.
    View in: PubMed
    Score: 0.086
  8. Opposing Actions of Fibroblast and Cardiomyocyte Smad3 Signaling in the Infarcted Myocardium. Circulation. 2018 02 13; 137(7):707-724.
    View in: PubMed
    Score: 0.078
  9. Tissue transglutaminase induction in the pressure-overloaded myocardium regulates matrix remodelling. Cardiovasc Res. 2017 Jul 01; 113(8):892-905.
    View in: PubMed
    Score: 0.076
  10. Myocardial Galectin-3 Expression Is Associated with Remodeling of the Pressure-Overloaded Heart and May Delay the Hypertrophic Response without Affecting Survival, Dysfunction, and Cardiac Fibrosis. Am J Pathol. 2016 05; 186(5):1114-27.
    View in: PubMed
    Score: 0.069
  11. The role of Interleukin Receptor Associated Kinase (IRAK)-M in regulation of myofibroblast phenotype in vitro, and in an experimental model of non-reperfused myocardial infarction. J Mol Cell Cardiol. 2015 Dec; 89(Pt B):223-31.
    View in: PubMed
    Score: 0.068
  12. Smad3 Signaling Promotes Fibrosis While Preserving Cardiac and Aortic Geometry in Obese Diabetic Mice. Circ Heart Fail. 2015 Jul; 8(4):788-98.
    View in: PubMed
    Score: 0.065
  13. Thrombospondin-1 induction in the diabetic myocardium stabilizes the cardiac matrix in addition to promoting vascular rarefaction through angiopoietin-2 upregulation. Circ Res. 2013 Dec 06; 113(12):1331-44.
    View in: PubMed
    Score: 0.058
  14. IL-1 induces proinflammatory leukocyte infiltration and regulates fibroblast phenotype in the infarcted myocardium. J Immunol. 2013 Nov 01; 191(9):4838-48.
    View in: PubMed
    Score: 0.058
  15. Lack of specificity of fibroblast-specific protein 1 in cardiac remodeling and fibrosis. Am J Physiol Heart Circ Physiol. 2013 Nov 01; 305(9):H1363-72.
    View in: PubMed
    Score: 0.058
  16. Thrombospondin-1 regulates adiposity and metabolic dysfunction in diet-induced obesity enhancing adipose inflammation and stimulating adipocyte proliferation. Am J Physiol Endocrinol Metab. 2013 Aug 01; 305(3):E439-50.
    View in: PubMed
    Score: 0.057
  17. Endogenous IRAK-M attenuates postinfarction remodeling through effects on macrophages and fibroblasts. Arterioscler Thromb Vasc Biol. 2012 Nov; 32(11):2598-608.
    View in: PubMed
    Score: 0.054
  18. Endogenous thrombospondin 1 protects the pressure-overloaded myocardium by modulating fibroblast phenotype and matrix metabolism. Hypertension. 2011 Nov; 58(5):902-11.
    View in: PubMed
    Score: 0.051
  19. The cellular specificity of leptin-mediated actions in the infarcted heart. Cardiovasc Res. 2011 Jan 01; 89(1):9-11.
    View in: PubMed
    Score: 0.048
  20. Smad3 signaling critically regulates fibroblast phenotype and function in healing myocardial infarction. Circ Res. 2010 Aug 06; 107(3):418-28.
    View in: PubMed
    Score: 0.046
  21. Induction of the CXC chemokine interferon-gamma-inducible protein 10 regulates the reparative response following myocardial infarction. Circ Res. 2009 Nov 06; 105(10):973-83.
    View in: PubMed
    Score: 0.044
  22. CD44 is critically involved in infarct healing by regulating the inflammatory and fibrotic response. J Immunol. 2008 Feb 15; 180(4):2625-33.
    View in: PubMed
    Score: 0.040
  23. Essential role of Smad3 in infarct healing and in the pathogenesis of cardiac remodeling. Circulation. 2007 Nov 06; 116(19):2127-38.
    View in: PubMed
    Score: 0.039
  24. Critical role of monocyte chemoattractant protein-1/CC chemokine ligand 2 in the pathogenesis of ischemic cardiomyopathy. Circulation. 2007 Feb 06; 115(5):584-92.
    View in: PubMed
    Score: 0.037
  25. Interleukin-10 is not a critical regulator of infarct healing and left ventricular remodeling. Cardiovasc Res. 2007 May 01; 74(2):313-22.
    View in: PubMed
    Score: 0.036
  26. Critical role of endogenous thrombospondin-1 in preventing expansion of healing myocardial infarcts. Circulation. 2005 Jun 07; 111(22):2935-42.
    View in: PubMed
    Score: 0.033
  27. CCL2/Monocyte Chemoattractant Protein-1 regulates inflammatory responses critical to healing myocardial infarcts. Circ Res. 2005 Apr 29; 96(8):881-9.
    View in: PubMed
    Score: 0.032
  28. Smad7 effects on TGF-? and ErbB2 restrain myofibroblast activation and protect from postinfarction heart failure. J Clin Invest. 2022 02 01; 132(3).
    View in: PubMed
    Score: 0.026
  29. Uncontrolled angiogenic precursor expansion causes coronary artery anomalies in mice lacking Pofut1. Nat Commun. 2017 09 18; 8(1):578.
    View in: PubMed
    Score: 0.019
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.