Connection

EDWARD COOPER to Humans

This is a "connection" page, showing publications EDWARD COOPER has written about Humans.
Connection Strength

0.159
  1. Plural molecular and cellular mechanisms of pore domain KCNQ2 encephalopathy. Elife. 2025 Jan 06; 13.
    View in: PubMed
    Score: 0.027
  2. KCNQ channel PIP2 modulation: Two loose links, three rings, and a twist. Neuron. 2022 01 19; 110(2):178-180.
    View in: PubMed
    Score: 0.022
  3. An Ankyrin-G N-terminal Gate and Protein Kinase CK2 Dually Regulate Binding of Voltage-gated Sodium and KCNQ2/3 Potassium Channels. J Biol Chem. 2015 Jul 03; 290(27):16619-32.
    View in: PubMed
    Score: 0.014
  4. Anaortic off-pump coronary artery bypass grafting in the elderly and very elderly. Heart Lung Circ. 2013 Dec; 22(12):989-95.
    View in: PubMed
    Score: 0.012
  5. Made for "anchorin": Kv7.2/7.3 (KCNQ2/KCNQ3) channels and the modulation of neuronal excitability in vertebrate axons. Semin Cell Dev Biol. 2011 Apr; 22(2):185-92.
    View in: PubMed
    Score: 0.010
  6. Ion channel clustering at the axon initial segment and node of Ranvier evolved sequentially in early chordates. PLoS Genet. 2008 Dec; 4(12):e1000317.
    View in: PubMed
    Score: 0.009
  7. A common ankyrin-G-based mechanism retains KCNQ and NaV channels at electrically active domains of the axon. J Neurosci. 2006 Mar 08; 26(10):2599-613.
    View in: PubMed
    Score: 0.007
  8. Identification by mass spectrometry and functional characterization of two phosphorylation sites of KCNQ2/KCNQ3 channels. Proc Natl Acad Sci U S A. 2005 Dec 06; 102(49):17828-33.
    View in: PubMed
    Score: 0.007
  9. Phenotypic and functional assessment of two novel KCNQ2 gain-of-function variants Y141N and G239S and effects of amitriptyline treatment. Neurotherapeutics. 2024 Jan; 21(1):e00296.
    View in: PubMed
    Score: 0.006
  10. M-channels: neurological diseases, neuromodulation, and drug development. Arch Neurol. 2003 Apr; 60(4):496-500.
    View in: PubMed
    Score: 0.006
  11. KCNQ2 R144 variants cause neurodevelopmental disability with language impairment and autistic features without neonatal seizures through a gain-of-function mechanism. EBioMedicine. 2022 Jul; 81:104130.
    View in: PubMed
    Score: 0.006
  12. High-throughput evaluation of epilepsy-associated KCNQ2 variants reveals functional and pharmacological heterogeneity. JCI Insight. 2022 03 08; 7(5).
    View in: PubMed
    Score: 0.005
  13. Characteristics of KCNQ2 variants causing either benign neonatal epilepsy or developmental and epileptic encephalopathy. Epilepsia. 2019 09; 60(9):1870-1880.
    View in: PubMed
    Score: 0.005
  14. Epileptic Encephalopathy In A Patient With A Novel Variant In The Kv7.2 S2 Transmembrane Segment: Clinical, Genetic, and Functional Features. Int J Mol Sci. 2019 Jul 10; 20(14).
    View in: PubMed
    Score: 0.005
  15. Autism and developmental disability caused by KCNQ3 gain-of-function variants. Ann Neurol. 2019 08; 86(2):181-192.
    View in: PubMed
    Score: 0.005
  16. Neonatal nonepileptic myoclonus is a prominent clinical feature of KCNQ2 gain-of-function variants R201C and R201H. Epilepsia. 2017 03; 58(3):436-445.
    View in: PubMed
    Score: 0.004
  17. Infantile spasms and encephalopathy without preceding neonatal seizures caused by KCNQ2 R198Q, a gain-of-function variant. Epilepsia. 2017 01; 58(1):e10-e15.
    View in: PubMed
    Score: 0.004
  18. A hierarchy of ankyrin-spectrin complexes clusters sodium channels at nodes of Ranvier. Nat Neurosci. 2014 Dec; 17(12):1664-72.
    View in: PubMed
    Score: 0.003
  19. Channel-anchored protein kinase CK2 and protein phosphatase 1 reciprocally regulate KCNQ2-containing M-channels via phosphorylation of calmodulin. J Biol Chem. 2014 Apr 18; 289(16):11536-11544.
    View in: PubMed
    Score: 0.003
  20. KCNQ2 is a nodal K+ channel. J Neurosci. 2004 Feb 04; 24(5):1236-44.
    View in: PubMed
    Score: 0.002
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.