Connection

ZHANDONG LIU to Disease Models, Animal

This is a "connection" page, showing publications ZHANDONG LIU has written about Disease Models, Animal.
Connection Strength

0.791
  1. A Portal to Visualize Transcriptome Profiles in Mouse Models of Neurological Disorders. Genes (Basel). 2019 09 26; 10(10).
    View in: PubMed
    Score: 0.322
  2. Apparent bias toward long gene misregulation in MeCP2 syndromes disappears after controlling for baseline variations. Nat Commun. 2018 08 13; 9(1):3225.
    View in: PubMed
    Score: 0.074
  3. Disease-related gene module detection based on a multi-label propagation clustering algorithm. PLoS One. 2017; 12(5):e0178006.
    View in: PubMed
    Score: 0.068
  4. A Comprehensive and Integrative Approach to MeCP2 Disease Transcriptomics. Int J Mol Sci. 2023 Mar 07; 24(6).
    View in: PubMed
    Score: 0.026
  5. MeCP2 regulates Gdf11, a dosage-sensitive gene critical for neurological function. Elife. 2023 02 27; 12.
    View in: PubMed
    Score: 0.025
  6. Disruption of MeCP2-TCF20 complex underlies distinct neurodevelopmental disorders. Proc Natl Acad Sci U S A. 2022 01 25; 119(4).
    View in: PubMed
    Score: 0.024
  7. Downregulation of glial genes involved in synaptic function mitigates Huntington's disease pathogenesis. Elife. 2021 04 19; 10.
    View in: PubMed
    Score: 0.022
  8. The bone microenvironment increases phenotypic plasticity of ER+ breast cancer cells. Dev Cell. 2021 04 19; 56(8):1100-1117.e9.
    View in: PubMed
    Score: 0.022
  9. Meta-Analysis of the Alzheimer's Disease Human Brain Transcriptome and Functional Dissection in Mouse Models. Cell Rep. 2020 07 14; 32(2):107908.
    View in: PubMed
    Score: 0.021
  10. Type I interferon response drives neuroinflammation and synapse loss in Alzheimer disease. J Clin Invest. 2020 04 01; 130(4):1912-1930.
    View in: PubMed
    Score: 0.021
  11. Complement C3aR Inactivation Attenuates Tau Pathology and Reverses an Immune Network Deregulated in Tauopathy Models and Alzheimer's Disease. Neuron. 2018 12 19; 100(6):1337-1353.e5.
    View in: PubMed
    Score: 0.019
  12. TFEB enhances astroglial uptake of extracellular tau species and reduces tau spreading. J Exp Med. 2018 09 03; 215(9):2355-2377.
    View in: PubMed
    Score: 0.019
  13. High-Throughput Functional Analysis Distinguishes Pathogenic, Nonpathogenic, and Compensatory Transcriptional Changes in Neurodegeneration. Cell Syst. 2018 07 25; 7(1):28-40.e4.
    View in: PubMed
    Score: 0.018
  14. Otud7a Knockout Mice Recapitulate Many Neurological Features of 15q13.3 Microdeletion Syndrome. Am J Hum Genet. 2018 02 01; 102(2):296-308.
    View in: PubMed
    Score: 0.018
  15. Extensive cryptic splicing upon loss of RBM17 and TDP43 in neurodegeneration models. Hum Mol Genet. 2016 12 01; 25(23):5083-5093.
    View in: PubMed
    Score: 0.017
  16. Loss of MeCP2 in the rat models regression, impaired sociability and transcriptional deficits of Rett syndrome. Hum Mol Genet. 2016 08 01; 25(15):3284-3302.
    View in: PubMed
    Score: 0.016
  17. Reversal of phenotypes in MECP2 duplication mice using genetic rescue or antisense oligonucleotides. Nature. 2015 Dec 03; 528(7580):123-6.
    View in: PubMed
    Score: 0.015
  18. FOXG1 expression shows correlation with neuronal differentiation in cerebellar development, aggressive phenotype in medulloblastomas, and survival in a xenograft model of medulloblastoma. Hum Pathol. 2015 Dec; 46(12):1859-71.
    View in: PubMed
    Score: 0.015
  19. A drosophila genetic resource of mutants to study mechanisms underlying human genetic diseases. Cell. 2014 Sep 25; 159(1):200-214.
    View in: PubMed
    Score: 0.014
  20. Serine catabolism regulates mitochondrial redox control during hypoxia. Cancer Discov. 2014 Dec; 4(12):1406-17.
    View in: PubMed
    Score: 0.014
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.