Connection

HELEN PIWNICA-WORMS to Checkpoint Kinase 1

This is a "connection" page, showing publications HELEN PIWNICA-WORMS has written about Checkpoint Kinase 1.
Connection Strength

1.229
  1. CDKN2A/p16 Deletion in Head and Neck Cancer Cells Is Associated with CDK2 Activation, Replication Stress, and Vulnerability to CHK1 Inhibition. Cancer Res. 2018 02 01; 78(3):781-797.
    View in: PubMed
    Score: 0.142
  2. CHK1 Inhibition in Small-Cell Lung Cancer Produces Single-Agent Activity in Biomarker-Defined Disease Subsets and Combination Activity with Cisplatin or Olaparib. Cancer Res. 2017 07 15; 77(14):3870-3884.
    View in: PubMed
    Score: 0.136
  3. CRL4(CDT2) targets CHK1 for PCNA-independent destruction. Mol Cell Biol. 2013 Jan; 33(2):213-26.
    View in: PubMed
    Score: 0.099
  4. Targeting Chk1 in p53-deficient triple-negative breast cancer is therapeutically beneficial in human-in-mouse tumor models. J Clin Invest. 2012 Apr; 122(4):1541-52.
    View in: PubMed
    Score: 0.095
  5. Synthetic lethality of Chk1 inhibition combined with p53 and/or p21 loss during a DNA damage response in normal and tumor cells. Oncogene. 2013 Jan 31; 32(5):577-88.
    View in: PubMed
    Score: 0.095
  6. Death by releasing the breaks: CHK1 inhibitors as cancer therapeutics. Trends Mol Med. 2011 Feb; 17(2):88-96.
    View in: PubMed
    Score: 0.087
  7. DDB1 targets Chk1 to the Cul4 E3 ligase complex in normal cycling cells and in cells experiencing replication stress. Cancer Res. 2009 Mar 15; 69(6):2630-7.
    View in: PubMed
    Score: 0.077
  8. GSK-3 beta targets Cdc25A for ubiquitin-mediated proteolysis, and GSK-3 beta inactivation correlates with Cdc25A overproduction in human cancers. Cancer Cell. 2008 Jan; 13(1):36-47.
    View in: PubMed
    Score: 0.071
  9. Phosphorylation of Chk1 by ATR is antagonized by a Chk1-regulated protein phosphatase 2A circuit. Mol Cell Biol. 2006 Oct; 26(20):7529-38.
    View in: PubMed
    Score: 0.065
  10. Chk1 kinase negatively regulates mitotic function of Cdc25A phosphatase through 14-3-3 binding. Mol Cell Biol. 2003 Nov; 23(21):7488-97.
    View in: PubMed
    Score: 0.053
  11. Cdc25A regulation: to destroy or not to destroy--is that the only question? Cell Cycle. 2003 Sep-Oct; 2(5):455-7.
    View in: PubMed
    Score: 0.053
  12. Disruption of the checkpoint kinase 1/cell division cycle 25A pathway abrogates ionizing radiation-induced S and G2 checkpoints. Proc Natl Acad Sci U S A. 2002 Nov 12; 99(23):14795-800.
    View in: PubMed
    Score: 0.050
  13. ATR-mediated checkpoint pathways regulate phosphorylation and activation of human Chk1. Mol Cell Biol. 2001 Jul; 21(13):4129-39.
    View in: PubMed
    Score: 0.045
  14. The Chk1 protein kinase and the Cdc25C regulatory pathways are targets of the anticancer agent UCN-01. J Biol Chem. 2000 Feb 25; 275(8):5600-5.
    View in: PubMed
    Score: 0.041
  15. Ca2+-Stimulated AMPK-Dependent Phosphorylation of Exo1 Protects Stressed Replication Forks from Aberrant Resection. Mol Cell. 2019 06 20; 74(6):1123-1137.e6.
    View in: PubMed
    Score: 0.039
  16. Mitotic and G2 checkpoint control: regulation of 14-3-3 protein binding by phosphorylation of Cdc25C on serine-216. Science. 1997 Sep 05; 277(5331):1501-5.
    View in: PubMed
    Score: 0.035
  17. Lack of PTEN sequesters CHK1 and initiates genetic instability. Cancer Cell. 2005 Feb; 7(2):193-204.
    View in: PubMed
    Score: 0.015
  18. The human decatenation checkpoint. Proc Natl Acad Sci U S A. 2001 Oct 09; 98(21):12044-9.
    View in: PubMed
    Score: 0.012
  19. Replication checkpoint requires phosphorylation of the phosphatase Cdc25 by Cds1 or Chk1. Nature. 1998 Oct 01; 395(6701):507-10.
    View in: PubMed
    Score: 0.009
  20. Conservation of the Chk1 checkpoint pathway in mammals: linkage of DNA damage to Cdk regulation through Cdc25. Science. 1997 Sep 05; 277(5331):1497-501.
    View in: PubMed
    Score: 0.009
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.