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David Hon Quach

InstitutionBaylor College of Medicine
DepartmentDepartment of Medicine
DivisionMedicine-General Medicine
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    Other Positions
    InstitutionBaylor College of Medicine
    DepartmentCenter for Cell & Gene Therapy
    DivisionCenter for Cell & Gene Therapy

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    F31NS066721     (QUACH, DAVID HON)Apr 1, 2010 - Mar 31, 2013
    Transcriptional regulation by Egr3 in sympathetic nervous system development
    Role: Principal Investigator

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    Publications listed below are automatically derived from MEDLINE/PubMed and other sources, which might result in incorrect or missing publications. Faculty can login to make corrections and additions.
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    1. Quach DH, Becerra-Dominguez L, Rouce RH, Rooney CM. A strategy to protect off-the-shelf cell therapy products using virus-specific T-cells engineered to eliminate alloreactive T-cells. J Transl Med. 2019 07 24; 17(1):240. PMID: 31340822.
      View in: PubMed
    2. Quach DH, Oliveira-Fernandes M, Gruner KA, Tourtellotte WG. A sympathetic neuron autonomous role for Egr3-mediated gene regulation in dendrite morphogenesis and target tissue innervation. J Neurosci. 2013 Mar 06; 33(10):4570-83. PMID: 23467373.
      View in: PubMed
    3. Li L, Eldredge LC, Quach DH, Honasoge A, Gruner K, Tourtellotte WG. Egr3 dependent sympathetic target tissue innervation in the absence of neuron death. PLoS One. 2011; 6(9):e25696. PMID: 21980528.
      View in: PubMed
    4. Eldredge LC, Gao XM, Quach DH, Li L, Han X, Lomasney J, Tourtellotte WG. Abnormal sympathetic nervous system development and physiological dysautonomia in Egr3-deficient mice. Development. 2008 Sep; 135(17):2949-57. PMID: 18653557.
      View in: PubMed
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