Connection

PRADIP SAHA to Mice, Inbred C57BL

This is a "connection" page, showing publications PRADIP SAHA has written about Mice, Inbred C57BL.
Connection Strength

0.351
  1. Metabolic adaptations in the absence of perilipin: increased beta-oxidation and decreased hepatic glucose production associated with peripheral insulin resistance but normal glucose tolerance in perilipin-null mice. J Biol Chem. 2004 Aug 20; 279(34):35150-8.
    View in: PubMed
    Score: 0.030
  2. A novel model of gestational diabetes: Acute high fat high sugar diet results in insulin resistance and beta cell dysfunction during pregnancy in mice. PLoS One. 2022; 17(12):e0279041.
    View in: PubMed
    Score: 0.027
  3. PHDs/CPT1B/VDAC1 axis regulates long-chain fatty acid oxidation in cardiomyocytes. Cell Rep. 2021 10 05; 37(1):109767.
    View in: PubMed
    Score: 0.025
  4. Loss of bone morphogenetic protein-binding endothelial regulator causes insulin resistance. Nat Commun. 2021 03 26; 12(1):1927.
    View in: PubMed
    Score: 0.024
  5. STAT1 Dissociates Adipose Tissue Inflammation From Insulin Sensitivity in Obesity. Diabetes. 2020 12; 69(12):2630-2641.
    View in: PubMed
    Score: 0.024
  6. Metabolic dysregulation in the Atp7b-/- Wilson's disease mouse model. Proc Natl Acad Sci U S A. 2020 01 28; 117(4):2076-2083.
    View in: PubMed
    Score: 0.022
  7. Asprosin is a centrally acting orexigenic hormone. Nat Med. 2017 Dec; 23(12):1444-1453.
    View in: PubMed
    Score: 0.019
  8. Hepatic FXR/SHP axis modulates systemic glucose and fatty acid homeostasis in aged mice. Hepatology. 2017 08; 66(2):498-509.
    View in: PubMed
    Score: 0.019
  9. PLIN2 is a Key Regulator of the Unfolded Protein Response and Endoplasmic Reticulum Stress Resolution in Pancreatic ? Cells. Sci Rep. 2017 01 19; 7:40855.
    View in: PubMed
    Score: 0.018
  10. Asprosin, a Fasting-Induced Glucogenic Protein Hormone. Cell. 2016 Apr 21; 165(3):566-79.
    View in: PubMed
    Score: 0.017
  11. Proinsulin-producing, hyperglycemia-induced adipose tissue macrophages underlie insulin resistance in high fat-fed diabetic mice. FASEB J. 2015 Aug; 29(8):3537-48.
    View in: PubMed
    Score: 0.016
  12. Molecular mechanisms underlying fasting modulated liver insulin sensitivity and metabolism in male lipodystrophic Bscl2/Seipin-deficient mice. Endocrinology. 2014 Nov; 155(11):4215-25.
    View in: PubMed
    Score: 0.015
  13. A duplication CNV that conveys traits reciprocal to metabolic syndrome and protects against diet-induced obesity in mice and men. PLoS Genet. 2012; 8(5):e1002713.
    View in: PubMed
    Score: 0.013
  14. Carbohydrate response element-binding protein (ChREBP) plays a pivotal role in beta cell glucotoxicity. Diabetologia. 2012 Jun; 55(6):1783-96.
    View in: PubMed
    Score: 0.013
  15. The coactivator SRC-1 is an essential coordinator of hepatic glucose production. Cell Metab. 2010 Dec 01; 12(6):606-18.
    View in: PubMed
    Score: 0.012
  16. Targeted inactivation of MLL3 histone H3-Lys-4 methyltransferase activity in the mouse reveals vital roles for MLL3 in adipogenesis. Proc Natl Acad Sci U S A. 2008 Dec 09; 105(49):19229-34.
    View in: PubMed
    Score: 0.010
  17. Molecular characterization of the role of orphan receptor small heterodimer partner in development of fatty liver. Hepatology. 2007 Jul; 46(1):147-57.
    View in: PubMed
    Score: 0.009
  18. Orphan receptor small heterodimer partner is an important mediator of glucose homeostasis. Mol Endocrinol. 2006 Nov; 20(11):2671-81.
    View in: PubMed
    Score: 0.009
  19. Ablation of ghrelin improves the diabetic but not obese phenotype of ob/ob mice. Cell Metab. 2006 May; 3(5):379-86.
    View in: PubMed
    Score: 0.009
  20. Farnesoid X receptor is essential for normal glucose homeostasis. J Clin Invest. 2006 Apr; 116(4):1102-9.
    View in: PubMed
    Score: 0.009
  21. Reversal of obesity by targeted ablation of adipose tissue. Nat Med. 2004 Jun; 10(6):625-32.
    View in: PubMed
    Score: 0.008
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.