"Hyperalgesia" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
An increased sensation of pain or discomfort produced by mimimally noxious stimuli due to damage to soft tissue containing NOCICEPTORS or injury to a peripheral nerve.
| Descriptor ID |
D006930
|
| MeSH Number(s) |
C10.597.751.791.400 C23.888.592.763.770.400
|
| Concept/Terms |
Hyperalgesia- Hyperalgesia
- Hyperalgesias
- Hyperalgesic Sensations
Mechanical Allodynia- Mechanical Allodynia
- Allodynias, Mechanical
- Hyperalgesia, Mechanical
- Hyperalgesias, Mechanical
- Mechanical Hyperalgesia
- Allodynia, Mechanical
Tactile Allodynia- Tactile Allodynia
- Allodynias, Tactile
- Hyperalgesia, Tactile
- Hyperalgesias, Tactile
- Allodynia, Tactile
Hyperalgesia, Thermal- Hyperalgesia, Thermal
- Hyperalgesias, Thermal
- Thermal Hyperalgesia
- Thermal Allodynia
- Allodynia, Thermal
- Allodynias, Thermal
|
Below are MeSH descriptors whose meaning is more general than "Hyperalgesia".
Below are MeSH descriptors whose meaning is more specific than "Hyperalgesia".
This graph shows the total number of publications written about "Hyperalgesia" by people in this website by year, and whether "Hyperalgesia" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1997 | 1 | 0 | 1 |
| 2004 | 0 | 1 | 1 |
| 2005 | 2 | 0 | 2 |
| 2006 | 1 | 0 | 1 |
| 2007 | 1 | 0 | 1 |
| 2009 | 0 | 1 | 1 |
| 2010 | 1 | 0 | 1 |
| 2013 | 0 | 1 | 1 |
| 2014 | 0 | 1 | 1 |
| 2016 | 0 | 1 | 1 |
| 2017 | 0 | 1 | 1 |
| 2018 | 2 | 1 | 3 |
| 2019 | 1 | 0 | 1 |
| 2020 | 1 | 1 | 2 |
| 2021 | 1 | 0 | 1 |
| 2024 | 1 | 0 | 1 |
| 2026 | 1 | 0 | 1 |
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Below are the most recent publications written about "Hyperalgesia" by people in Profiles.
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Cell-specific role of histone deacetylase 6 in chemotherapy-induced mechanical allodynia and loss of intraepidermal nerve fibers. Pain. 2019 12; 160(12):2877-2890.
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The Cancer Chemotherapeutic Paclitaxel Increases Human and Rodent Sensory Neuron Responses to TRPV1 by Activation of TLR4. J Neurosci. 2015 Sep 30; 35(39):13487-500.
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The anti-diabetic drug metformin protects against chemotherapy-induced peripheral neuropathy in a mouse model. PLoS One. 2014; 9(6):e100701.
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Monocytes/Macrophages control resolution of transient inflammatory pain. J Pain. 2014 May; 15(5):496-506.
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A novel p38 MAPK docking-groove-targeted compound is a potent inhibitor of inflammatory hyperalgesia. Biochem J. 2014 May 01; 459(3):427-39.
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Role of ATP-sensitive potassium channels in modulating nociception in rat model of bone cancer pain. Brain Res. 2014 Mar 20; 1554:29-35.
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Balancing GRK2 and EPAC1 levels prevents and relieves chronic pain. J Clin Invest. 2013 Dec; 123(12):5023-34.
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Nerve injury increases GluA2-lacking AMPA receptor prevalence in spinal cords: functional significance and signaling mechanisms. J Pharmacol Exp Ther. 2013 Dec; 347(3):765-72.
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Electroacupuncture improves thermal and mechanical sensitivities in a rat model of postherpetic neuralgia. Mol Pain. 2013 Apr 03; 9:18.
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MicroRNA-124 as a novel treatment for persistent hyperalgesia. J Neuroinflammation. 2012 Jun 25; 9:143.